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ALS-causative mutations in FUS/TLS confer gain and loss of function by altered association with SMN and U1-snRNP

  • Author(s): Huang, Eric
  • Sun, S
  • Ling, SC
  • Qiu, J
  • Albuquerque, CP
  • Zhou, Y
  • Tokunaga, S
  • Li, H
  • Qiu, H
  • Bui, A
  • Yeo, GW
  • et al.

Published Web Location

http://europepmc.org/articles/PMC4338613?pdf=render
No data is associated with this publication.
Abstract

© 2015 Macmillan Publishers Limited. All rights reserved.The RNA-binding protein FUS/TLS, mutation in which is causative of the fatal motor neuron disease amyotrophic lateral sclerosis (ALS), is demonstrated to directly bind to the U1-snRNP and SMN complex

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