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ALS-causative mutations in FUS/TLS confer gain and loss of function by altered association with SMN and U1-snRNP
Published Web Location
http://europepmc.org/articles/PMC4338613?pdf=renderNo data is associated with this publication.
Abstract
© 2015 Macmillan Publishers Limited. All rights reserved.The RNA-binding protein FUS/TLS, mutation in which is causative of the fatal motor neuron disease amyotrophic lateral sclerosis (ALS), is demonstrated to directly bind to the U1-snRNP and SMN complex
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