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The Role of ATM in the Deficiency in Nonhomologous End-Joining near Telomeres in a Human Cancer Cell Line

  • Author(s): Murnane, John
  • Muraki, K
  • Han, L
  • Miller, D
  • Murnane, JP
  • et al.

Published Web Location

http://europepmc.org/articles/PMC3610639?pdf=render
No data is associated with this publication.
Abstract

Telomeres distinguish chromosome ends from double-strand breaks (DSBs) and prevent chromosome fusion. However, telomeres can also interfere with DNA repair, as shown by a deficiency in nonhomologous end joining (NHEJ) and an increase in large deletions at

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