Skip to main content
The Role of ATM in the Deficiency in Nonhomologous End-Joining near Telomeres in a Human Cancer Cell Line
Published Web Location
http://europepmc.org/articles/PMC3610639?pdf=renderNo data is associated with this publication.
Abstract
Telomeres distinguish chromosome ends from double-strand breaks (DSBs) and prevent chromosome fusion. However, telomeres can also interfere with DNA repair, as shown by a deficiency in nonhomologous end joining (NHEJ) and an increase in large deletions at
Many UC-authored scholarly publications are freely available on this site because of the UC's open access policies. Let us know how this access is important for you.