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The Ca2+-activated K+ channel KCa3.1 as a potential new target for the prevention of allograft vasculopathy
Published Web Location
http://europepmc.org/articles/PMC3843675?pdf=renderNo data is associated with this publication.
Abstract
Allograft vasculopathy (AV) remains one of the major challenges to the long-term functioning of solid organ transplants. Although its exact pathogenesis remains unclear, AV is characterized by both fibromuscular proliferation and infiltration of CD4+ memor
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