Skip to main content
Mechanisms for U2AF to define 3′ splice sites and regulate alternative splicing in the human genome
- Shao, Changwei;
- Yang, Bo;
- Wu, Tongbin;
- Huang, Jie;
- Tang, Peng;
- Zhou, Yu;
- Zhou, Jie;
- Qiu, Jinsong;
- Jiang, Li;
- Li, Hairi;
- Chen, Geng;
- Sun, Hui;
- Zhang, Yi;
- Denise, Alain;
- Zhang, Dong-Er;
- Fu, Xiang-Dong
Published Web Location
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4429597/No data is associated with this publication.
Abstract
U2AF is known to affect 3′-splice-site selection. Here, Fu and colleagues use genome-wide analysis of U2AF-RNA interactions to define U2AF's key roles in gene expression and regulated splicing in normal and disease states.
Many UC-authored scholarly publications are freely available on this site because of the UC's open access policies. Let us know how this access is important for you.