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Deficiency of TDAG51 protects against atherosclerosis by modulating apoptosis, cholesterol efflux, and peroxiredoxin-1 expression

  • Author(s): Malloy, Mary
  • Kane, John
  • Hossain, GS
  • Lynn, EG
  • Maclean, KN
  • Zhou, J
  • Dickhout, JG
  • Lhot́ak, S
  • Trigatti, B
  • Capone, J
  • Rho, J
  • Tang, D
  • et al.

Published Web Location

http://europepmc.org/articles/PMC3698773?pdf=render
No data is associated with this publication.
Abstract

Background: Apoptosis caused by endoplasmic reticulum (ER) stress contributes to atherothrombosis, the underlying cause of cardiovascular disease (CVD). T-cell death-associated gene 51 (TDAG51), a member of the pleckstrin homology-like domain gene family,

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