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The IgM receptor FcμR limits tonic BCR signaling by regulating expression of the IgM BCR

Abstract

FcμR serves as a receptor for soluble IgM. Baumgarth and colleagues show that intracellular FcμR constrains the surface expression of IgM. Lack of FcμR alters B cell populations and enhances autoantibody production. FcμR thereby serves as a critical regulator of B cell homeostasis.

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