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Heart repair by reprogramming non-myocytes with cardiac transcription factors.

Published Web Location

http://europepmc.org/articles/PMC3367390?pdf=render
No data is associated with this publication.
Abstract

The adult mammalian heart possesses little regenerative potential following injury. Fibrosis due to activation of cardiac fibroblasts impedes cardiac regeneration and contributes to loss of contractile function, pathological remodelling and susceptibility

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