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Heart repair by reprogramming non-myocytes with cardiac transcription factors.
Published Web Location
http://europepmc.org/articles/PMC3367390?pdf=renderNo data is associated with this publication.
Abstract
The adult mammalian heart possesses little regenerative potential following injury. Fibrosis due to activation of cardiac fibroblasts impedes cardiac regeneration and contributes to loss of contractile function, pathological remodelling and susceptibility
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